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Pathogenicity island-dependent effects of Helicobacter pylori on intracellular signal transduction in epithelial cells.
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The anti-inflammatory compound curcumin inhibits Neisseria gonorrhoeae-induced NF-kappaB signaling, release of pro-inflammatory cytokines/chemokines and attenuates adhesion in late infection.
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Up-regulation of cathepsin X in Helicobacter pylori gastritis and gastric cancer.
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Analysis of the type IV secretion system-dependent cell motility of Helicobacter pylori- infected epithelial cells.
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Curcumin blocks NF-kappaB and the motogenic response in Helicobacter pylori-infected epithelial cells.
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Helicobacter pylori-induced epithelial cell signalling in gastric carcinogenesis.
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Inhibition of p38 MAP kinase- and RICK/NF-kappaB-signaling suppresses inflammatory bowel disease.
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Prevention of gastric cancer by Helicobacter pylori eradication.
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Fission yeast COP9/signalosome suppresses cullin activity through recruitment of the deubiquitylating enzyme Ubp12p.
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Helicobacter pylori CagA protein targets the c-Met receptor and enhances the motogenic response.
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Helicobacter pylori stimulates host cyclooxygenase-2 gene transcription : critical importance of MEK/ERK-dependent activation of USF1/-2 and CREB transcription factors.
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Helicobacter pylori stimulates host vascular endothelial growth factor-A (vegf-A) gene expression via MEK/ERK-dependent activation of Sp1 and Sp3. . - Gedr. ersch. in: FASEB J. 18(2004)1, S. 218 - 220.
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The mitogen-activated protein kinase p38 : a new molecular target for anti-inflammatory therapy in Crohn's disease?.
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